ASTA XANTHIN – 100 ML

$26.00

Asta Xanthin is used for prevention and curative treatment of deseases.

Myopathy dyspnea

Stiff lamb disease

White muscle disease

Exudative myopathy of swine

Description

ASTA XANTHIN – 100 ML

Asta Xanthin is used for prevention and curative treatment of deseases.

Myopathy dyspnea

Stiff lamb disease

White muscle disease

Exudative myopathy of swine

Asta-Xanthin-ATP-Methyl-prednisolone-b12-Cantine-potasium-sodium-selenite-product-for-race-horseandcamelsupplies

astaxanthin energy

New presentation:

 

Asta-Xanthin-race-Energy-and-oxygen-horseandcamelsupplies.com

Old presentation

 

FORMULATION

ATP………………………………………………………… 200 mg

Anhydrous Sodium Selenite…………………. 50 mg

Methylprednisolone……………………………… 250 mg

L-magnesium bi-aspartate……………………1500 mg

Vitamin B1……………………………………………..25 mg

DN-Carnitine………………………………………….100 mg

astaxanthin energy

Exipient to 100 ml

RACING APPLICATIONS

  • Muscle support
  • Enhance performance
  • Increase endurance
  • Increase red blood cells production
  • Delays fatigue

DOSAGE AND ADMINISTRATION

Adult Horses: 20 ml (IV-IM-SC)

Young Horses: 10 ml (IV-IM-SC)

SUGGESTED DOSAGE AND ADMINISTRATION FOR RACING

Horses: 10 cc for 5 days before the race (IV-IM-SC)

Camels: 7.5 cc for 5 days before the race (IV-IM-SC)

astaxanthin energy

SKU: Astaxanthin Categories: Most Popular, Energy & Power, Endurance, Protectors & Recovery Tags: antiinflammatory, Asta, ATP, endurance, energy, methylprednisolone, pain, reliever, Xanthin
In this study, we investigated anti-dermatitic effects of Astaxanthin (AST) in phthalic anhydride (PA)-induced atopic dermatitis (AD) animal model as well as in vitro model. AD-like lesion was induced by the topical application of 5% PA to the dorsal skin or ear of Hos:HR-1 mouse. After AD induction, 100 μl of 1 mg/ml and 2 mg/ml of AST (10 μg or 20 μg/cm2 ) was spread on the dorsum of ear or back skin three times a week for four weeks. We evaluated dermatitis severity, histopathological changes and changes in protein expression by Western blotting for inducible nitric oxide synthase (iNOS), cyclooxygenase-2 (COX-2), and nuclear factor-κB (NF-κB) activity. We also measured tumor necrosis factor- α (TNF-α), interleukin-1β (IL-1β), interleukin-6 (IL-6), and immunoglobulinE (IgE) concentration in the blood of AD mice by enzyme-linked immunosorbent assay (ELISA). AST treatment attenuated the development of PA-induced AD. Histological analysis showed that AST inhibited hyperkeratosis, mast cells and infiltration of inflammatory cells. AST treatment inhibited expression of iNOS and COX-2, and NF-κB activity as well as release of TNF-α, IL-1β, IL-6, and IgE. In addition, AST (5, 10, and 20μM) potently inhibited lipopolysaccharide (LPS) (1 μg/ml)-induced nitric oxide (NO) production, expression of iNOS and COX-2, and NF-κB DNA binding activities in RAW 264.7 macrophage cells. Our data demonstrated that AST could be a promising agent for AD by inhibition of NF-κB signaling. This article is protected by copyright. All rights reserved.

ASTA XANTHIN – 100 ML

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